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I Am More Than Just A Broken Pancreas & A Human Pincushion...

Kelly's intimate knowledge of living with diabetes makes her the perfect person to poke fun at all of our little eccentricities. If laughter really is the best medicine, then Kelly should be nominated for Sturgeon General."
Gary Scheiner - Certified Diabetes Educator, Owner & Operator of Integrated Diabetes Services, Author of "You Can Control Diabetes" and "How to Think Like A Pancreas."
Marx Brothers Fan for life, T1 for 20 years .

"Kelly Kunik performed her Diabetes Comedy Act at the
Diabetes, Exercise, & Sports Association (DESA) National meeting in Colorado Springs in June of 2007. She had the room laughing all night! It was great to see the light side of Diabetes for once...."

Rick Philbin, Type 1, Board of Directors, DESA

"Kelly was very engaging with her humor and positive attitude in looking at life with Diabetes on a lighter side. Everyone in my Diabetes Support group lowered their glucose levels with laughter that evening!"
Bryony Crane, RD CDE
Virtua Diabetes and Nutrition Svs

"Dr. Kelly keeps you laughing.......Great bedside manner!"
Boston Charlie - T1 30 years

"As a Diabetes Educator, I'm always looking for new ways to help patients. Kelly Kunik offers a unique way of educating patients through laughter.
There's a tremendous validation in Kelly's approach - Everybody thinks that no one once else has diabetes related issues, day & day out. Whether it getting your tubing caught on a door nob; acting out with a low blood sugar, or dealing with the same old questions. Silly or serious, Kelly's observations allow patients to feel better about themselves.
When people feel good about themselves, they practice better
self management. IT'S ALL GOOD.
We all had so much fun the night Kelly spoke to my Type 1 support group."

Cheryl Marco, RD, CDE
Thomas Jefferson University
Division of Endocrinology and Metabolic Diseases

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I'm getting Bald – Find Out What Causes Hair Loss and and get Effective and Natural Treatment

Many people suffer from hair loss & are worried about the cause of baldness. You are not alone if you are having a difficult time finding a balding treatment. Because in order to find an effective baldness remedy, it is important to know what causes hair loss first.

One major cause of baldness is genetics. However, hormones also play a massive part in hair loss & usually caused by hormonal imbalance.

For instance, men tend to make more androgens which affects the hair follicles thus resulting to baldness or thinning hair. For women, hair loss usually happens after the period of menopause where the levels of estrogen is low in production.

Other causes are excessive anxiety, stress, shock & physical stress from chemotherapy or surgery. In case of severe disease, the overall condition of the hair can be affected & result to poor health of the hair.

Tight scalp happens to people who are experiencing starvation when the circulation of blood is not healthy. The result -- a deficiency of natural oil of the hair leading to baldness or hair loss.

Weakened scalp circulation is & a cause of baldness. Tight scalp tends to affect hair follicles.

There is lovely news. Natural ingredients found in a balding treatment have been proven safe & effective.

For women, taking birth control medications can cause hormonal imbalance that leads to thinning hair. Definite diseases such as diabetes are known to be a cause of baldness & even scalp infections.

Natural herb extracts work in synergy to block DHT, boost your circulation & provide nutritional support. A few natural nutrients include Saw Palmetto, Nettle Root & Muira Puama.

& one of the best ingredients is a topical medication approved by the FDA, for both male & female hairloss, is called Minoxidil. This substance doesn't grow new hair, bit it does block the DHT & prolong the growth phase of hair. Because it provides more time for hair to grow out, you'll see a your full head of hair once again.

Also playing an important role are vitamins & minerals like vitamin B6, biotin, zinc & magnesium. Providing nutrition, these nutrients help support natural healthy hair growth and improve your body's health.

Now that you know the cause of baldness, if you are dealing with thinning hair recall that there is an effective & natural balding treatment. It is not an overnight balding cure but if you stick with it over the months you'll gradually see a full head of hair.

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New Type of Diabetes Drug Drops Weight With Blood Sugar

SGLT2 Inhibitors Boost Metformin, Can Be Used Even in Late-Stage Diabetes

By Daniel J. DeNoon
from WebMD
Now the first of these so-called SGLT2 inhibitors has been tested in a phase III clinical trial. It's dapagliflozin, being jointly developed by Bristol-Myers Squibb and AstraZeneca.

Study leader Clifford J. Bailey, PhD, is professor of clinical science at Aston University in Birmingham, England. "It works through an entirely different mechanism than any other diabetes drugs currently available," Bailey tells WebMD. "And you can add it on to other treatments and get an additional benefit. Plus as far as we can see, it can be used at any stage in the disease process."

And that's not all. Because dapagliflozin makes the body excrete excess sugar, it makes diabetes patients lose weight. Metformin helps patients lose weight, too, but those adding dapagliflozin to metformin lost about 4 and 1/2 more pounds than those taking metformin alone in the 24-week study.

The lost weight was not just water. Patients taking dapagliflozin had smaller waistlines, so the lost weight appears to have been fat.

Might this new diabetes drug work as a weight loss pill? No, says Bailey.

"The weight loss effect of the drug becomes less and less as the blood-sugar level comes to near normal," he notes. "Therefore the weight loss potential of this drug is very small at normal blood-sugar levels."

Weight loss isn't dapagliflozin's only extra benefit. It lowers blood pressure, too. Another plus is that the oral medication is taken only once a day. And the drug does not lower blood sugar to dangerously low levels -- yet another plus.

On the down side, patients taking dapagliflozin had an increased risk of genital infections. People with diabetes already are at higher risk of genital and urinary infections. Dapagliflozin may increase this risk.

And because the drug changes the way the body excretes fluid, there may be as-yet-unseen long-term consequences, suggests Luigi F. Meneghini, MD, MBA, director of the University of Miami's diabetes treatment center. Meneghini was not involved in the Bailey study.

"If you have more sugar in your urinary tract, you have more risk of infection. This probably has to do with people with diabetes and high blood sugar having more fungal infections," Meneghini tells WebMD. "That said, if your doctor is careful in monitoring for you infections and treating them when they occur, this is still a drug with a relatively low side-effect profile. But we need to see data on lots more people exposed to the drug for a longer time."

The Bailey study enrolled patients whose blood sugar was not adequately controlled by metformin alone. Their blood sugar was too high but not wildly out of control.

Sources include:

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Sometimes I feel like I'm on a different planet...

I was in a coffee shop reading for school. I was reading an article about patients writing illness narratives. It was arguing that such stories can be seen as a political act against a biomedical model that sees patient merely as disease and not as an whole being. I have issues with some of what the authors argue. I don't really see one nice neat narrative in illness. It's more like different stories of good days and bad days and, while, there might be a beginning, for people with chronic illness there is no end, or at least not in life.

When I got toward the end of paper I started to tear up... which was kind of weird, because I still wasn't into the article. By the end it argues that nurses are in a position to dialogue with patients and create a sort of co-constructed narrative of illness that suits both the practictioner and the patient. Fair enough, I guess, but I just didn't find it incredibly mindblowing. So, I couldn't really figure out why I was suddenly all choked up...

And then I realized that this was playing in the background:

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Huge Metastudy: "Non Diabetic" Blood Sugars Cause "Diabetic" Retionopathy

I have already documented on my main web site data proving that blood sugar levels considerably lower than those labeled "diabetic" produce changes in the retina leading to blindness.

Now this finding has been confirmed and quantified in a meta study that looked at records of "44,623 participants aged 20 to 79 years with gradable retinal photographs" which examined the correlations between signs of retinopathy and the subjects' fasting, 2 hour glucose tolerance test, and A1c results.

Glycemic Thresholds for Diabetes-Specific Retinopathy: Implications for Diagnostic Criteria for Diabetes:The DETECT-2 Collaboration Writing group. Stephen Colagiuri et al. Diabetes Care Published online before print October 26, 2010, doi: 10.2337/dc10-1206

The conclusion of the study was this:

A narrow threshold range for diabetes-specific retinopathy was identified for FPG and HbA1c but not for 2-h PG. The combined analyses suggest that the current diabetes diagnostic level for FPG could be lowered to 6.5 mmol/L [117 mg/dl] and that an HbA1c of 6.5% is a suitable alternative diagnostic criterion.
The metastudy found that "glycemic thresholds for diabetes-specific retinopathy were observed over the range 6.4-6.8 mmol/L [115 - 122 mg/dl] for F[asting]P[lasma]G[lucose] 9.8-10.6 mmol/L [176.4 - 191 mg/dl] for 2-h PG, and 6.3-6.7% for HbA1c.

From this we can safely conclude that "diabetic" retinopathy is indeed occurring at levels significantly below those established by the American Diabetes Association as defining diabetes.

Based on this, you should consider yourself at risk for retinal damage if you have fasting blood sugar over 115 mg/dl (6.4 mmol/L) , a 2 hour glucose tolerance test reading over 176 mg/dl [9.8 mmol/L) or an A1c over 6.3%.

However, you need to assess this information in light of the fact that retinopathy is a relatively late diabetic complication. Diabetic neuropathy--the nerve damage that leads to impotence, amputation, and autonomic dysfunction which raises blood pressure etc.--starts to become more common when 2 hour glucose tolerance test values go over 140 mg/dl, though there does not appear to be a direct correlation with A1c or fasting plasma glucose at the lower end of the range.

Heart disease incidence correlates with post-meal readings over 155 mg/dl and rises in a straight line from 4.7% A1cs becoming quite significant over 6%.

The good news is that though these values correlate with significant retinopathy in populations who follow traditional medical advice, keeping your blood sugar values under these thresholds after diagnosis using the strategy you will read HERE will keep you from developing it if you don't already have it, and even if you do, long term will give you a much better outcome.

You can read more about Diabetic Retinopathy HERE.

NOTE: In case you wonder why the ADA diagnostic criteria are so much higher than the levels at which diabetic retinopathy occurs, the answer is that the ADA set their diagnostic criteria years ago using on data from a few small non-European populations (Pima Native Americans and Pacific Islanders) whose diabetes is related to different genetic profiles and follows a different pattern from those common in European populations.

They did this on purpose out of a misguided desire to avoid diagnosing people with diabetes for as long as possible and have fought hard in the intervening decades to keep these flawed diagnostic criteria even though they have all been found woefully inadequate by a ton of research.

The whole sad history of how the ADA has worked for decades to ensure that you will have developed diabetic complications long before you are diagnosed with diabetes can be read HERE.

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The Link Between Diet Soda Consumption and Stroke

A study to be presented at the International Stroke Conference 2011 in Los Angeles is getting some play in the health news. You can read a good summary here:

U.S News and World Report: Can Diet Soda Boost Your Stroke Risk?

The researchers "evaluated the soda habits of 2,564 people enrolled in the large Northern Manhattan Study (NOMAS) to see if there was an association, if any, with stroke. The participants were 69 years of age, on average, and completed food questionnaires about the type of soda they drank and how often."

Over 9 years, 22% of the study subjects had a stroke. After controlling for age, gender, ethnicity, physical activity, calorie intake, smoking, alcohol drinking habits, the presence of metabolic syndrome, vascular disease in the limbs and heart disease history those who reported drinking diet soda as opposed to no soda were 48 percent more likely to have a stroke.

In another article covering this same story published by MSNBC
other doctors are quoted as suggesting the problem might be what they eat with the soda--fast food, or possibly something in caramel coloring used to give sodas a brown color, which has been linked to stroke in animal studies.

Alert readers of this blog will, however, remember another equally likely explanation--one that has been known for years, but has received no coverage in the press, because the media get too much advertising money from Coke and Pepsi.

The link between diet soda and stroke may well be the phosphoric acid that is used in the all brown-colored sodas. As documented in my earlier blog post Coke Adds Death, brown-colored sodas are known to damage the kidneys. In fact, drinking as few as two brown-colored sodas--either diet or regular--a day doubles the risk of developing chronic kidney disease. Researchers believe the phosphoric acid is the culprit. Phosphates are a known problem for people who already have kidney disease.

It turns out that kidney damage and cardiovascular disease are tightly linked, and the presence of kidney disease often points to the existence of other vascular problems. This makes it very possible that damage to the kidney from phosporic acid is contributing to vascular damage in the brain which leads to stroke.

If phosphoric acid is the problem--and it is likely, since people consuming non-brown colored sodas had a normal risk of chronic kidney disesae, you can avoid it by avoiding heavily-advertised brown sodas like Coke, Pepsi and Dr. Pepper, in favor of the light and colored sodas that don't. If in doubt read the label. If it says "phosporic acid" give it a miss. People with diabetes have enough issues to contend with kidney-wise without adding to them.


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